| dc.creator | World conference on lauric oils:sources, processing and applications
1994.
53762 | |
| dc.creator | Dayrit, Conrado S.
53763 | |
| dc.date | 1994. | |
| dc.identifier | URN:ISBN:0935315-56-X | |
| dc.description | Coconut oil, a saturated fat, and the saturared, animal-derived fats have been labeled unhealthy. The saturared fatty acis of coconut oil are predominantly medium-chain length (C6-C12), with absorption, transport, metabolim, and distribution characteristics radically different from those of the long-chain fats. Coconut oil-feeding experiments in various animal species, particularly rabbits, have been falwed by the nonphysiologic amounts of coconut oil given toa these animals and the failure to supplement the diet with essential oils, leading to essential fatty acid deficiecy. To date, no evidence has been presented to indicate that coconut oil causes coronary heart disease in human subjects or in populations that consume large amounts in their diets. On the contrary, the Polynesian study shwoed that the people of Puka Puka and Tokelau had normal serum cholesterol and very low incidence of coronary heart disease even thought they received 35-55% of their calories from fat, mostly from coconut and fish. A controlled, randomized clinical trial on high fat-consuming (37% of total calories) Causasian subjects fiven coconut oil as high as 60% of their daily fat intake showed no significant increase in total cholesterol and some increase in HDL whgile lowering the cholesterol: HDL ratio towards a more favorable ratio. Recent findings on atherogenesis explain why the lipid-cholesterol-atherogenesis theory is too simplistic and fails to explain many observations on coronary heart disease and cerebrovascular disease occurrence. | |
| dc.description | Incluye 27 referencias bibliográficas. | |
| dc.description | Coconut oil, a saturated fat, and the saturared, animal-derived fats have been labeled unhealthy. The saturared fatty acis of coconut oil are predominantly medium-chain length (C6-C12), with absorption, transport, metabolim, and distribution characteristics radically different from those of the long-chain fats. Coconut oil-feeding experiments in various animal species, particularly rabbits, have been falwed by the nonphysiologic amounts of coconut oil given toa these animals and the failure to supplement the diet with essential oils, leading to essential fatty acid deficiecy. To date, no evidence has been presented to indicate that coconut oil causes coronary heart disease in human subjects or in populations that consume large amounts in their diets. On the contrary, the Polynesian study shwoed that the people of Puka Puka and Tokelau had normal serum cholesterol and very low incidence of coronary heart disease even thought they received 35-55% of their calories from fat, mostly from coconut and fish. A controlled, randomized clinical trial on high fat-consuming (37% of total calories) Causasian subjects fiven coconut oil as high as 60% of their daily fat intake showed no significant increase in total cholesterol and some increase in HDL whgile lowering the cholesterol: HDL ratio towards a more favorable ratio. Recent findings on atherogenesis explain why the lipid-cholesterol-atherogenesis theory is too simplistic and fails to explain many observations on coronary heart disease and cerebrovascular disease occurrence. | |
| dc.language | ng | |
| dc.publisher | Champaign : AOCS, | |
| dc.subject | Aceite de coco. | |
| dc.subject | Colesterol. | |
| dc.subject | Enfermedades coronarias | |
| dc.subject | Salud humana | |
| dc.title | Health aspects of coconut oil. | |
| dc.type | text | |
